Zhanyang Qian | Neuroscience | Research Excellence Award

Dr. Zhanyang Qian | Neuroscience | Research Excellence Award

Nantong University | China

Dr. Zhanyang Qian is a neural regeneration scientist specializing in innovative therapeutic strategies for spinal cord injury, with a strong focus on immunomodulation, neuroinflammation, and cellular repair mechanisms. His research integrates advanced molecular immunology, mitochondrial biology, and translational animal models to uncover pathways that drive neural protection and functional recovery. He has secured multiple national-level grants and prestigious provincial talent recognitions, reflecting his rapidly rising impact in the field. Dr. Qian has authored a robust portfolio of high-impact publications, including several first- and corresponding-author papers in top-tier journals, with his work cited by leading neuroscience outlets. His studies have advanced understanding of microglial and astrocytic responses, efferocytosis in senescent macrophages, and mitophagy-driven neuroprotection, contributing valuable insights to the development of targeted interventions for central nervous system injury. Beyond research, he contributes to the scientific community as a committee member in neurological regeneration, serves on an editorial board, and reviews for reputable journals. Dr. Qian is recognized for his leadership in pioneering mechanistic discoveries and translating them toward clinically relevant therapeutic strategies, positioning him as an emerging expert in spinal cord injury repair and neural regeneration.

Profiles: Scopus | Orcid

Featured Publications

Xia, M., Li, C., Zhang, Y., Wang, T., Zhang, C., Zhou, J., Zhu, X., Hong, H., Li, H., Qian, Z., et al. (2025). GLP-1R activation restores Gas6-driven efferocytosis in senescent foamy macrophages to promote neural repair. Redox Biology, 103857.

Xia, M., Li, C., Chen, J., Wu, C., Zhang, J., Hong, H., Jiang, J., Xu, G., Qian, Z., & Cui, Z. (2025). Activation of FANCC attenuates mitochondrial ROS-driven necroptosis by targeting TBK1-dependent mitophagy in astrocytes after spinal cord injury. Theranostics.

Qian, Z., Li, R., Zhao, T., Xie, K., Li, P., Li, G., Shen, N., Gong, J., Hong, X., & Yang, L., et al. (2024). Blockade of the ADAM8–Fra-1 complex attenuates neuroinflammation by suppressing the Map3k4/MAPKs axis after spinal cord injury. Cellular & Molecular Biology Letters, 29, Article 589.

Qian, Z., Xia, M., Zhao, T., Li, Y., Li, G., Zhang, Y., Li, H., & Yang, L. (2024). ACOD1, rather than itaconate, facilitates p62-mediated activation of Nrf2 in microglia post spinal cord contusion. Clinical and Translational Medicine, 14, Article e1661.

Chang, J., Qian, Z., Wang, B., Cao, J., Zhang, S., Jiang, F., Kong, R., Yu, X., Cao, X., & Yang, L., et al. (2023). Transplantation of A2 type astrocytes promotes neural repair and remyelination after spinal cord injury. Cell Communication and Signaling, 21(1).

Yuxin Ma | Neuroscience | Best Researcher Award

Dr. Yuxin Ma | Neuroscience | Best Researcher Award

DR at Guangdong Pharmaceutical University, China

Dr. Yuxin Ma is a neuroscientist affiliated with Guangdong Pharmaceutical University. Her research focuses on neuropharmacology, particularly the effects of natural compounds on pain mechanisms and their affective comorbidities. She has made significant contributions to understanding the neural pathways involved in pain processing and has authored a high-impact study on the analgesic effects of astragalin.

Publication ProfileΒ 

Scopus

Educational Background πŸŽ“

  • Institution: Guangdong Pharmaceutical University

  • Field of Study: Neuroscience

Professional Experience πŸ’Ό

  • Designation: Researcher at Guangdong Pharmaceutical University

  • Key Responsibilities: Leading research on neuropharmacology, particularly investigating the therapeutic effects of natural compounds in pain modulation.

Research Interests πŸ”¬

  • Neuropharmacology of natural compounds

  • Neural mechanisms underlying pain and associated affective disorders

  • Autophagy and neuronal excitability in pain pathways

Conclusion🌟

Dr. Yuxin Ma’s research significantly advances the understanding of pain modulation through natural compounds. Her work on astragalin provides groundbreaking insights into its dual role in inhibiting neuronal excitability and promoting autophagy, paving the way for novel neurotherapeutic approaches. She remains committed to expanding the field of neuroscience and neuropharmacology through innovative research and collaboration.

Publications πŸ“š

πŸ“„ Article: Astragalin relieves inflammatory pain and negative mood in CFA mice by down-regulating mGluR5 signaling pathway
πŸ‘₯ Authors: R. Zhang, Runheng; J. Lin, Jiahong; S. Wang, Shuhan; … L. Zhang, Li; Y. Ma, Yuxin
πŸ“š Journal: Scientific Reports, 2025
πŸ”— Full text: 🚫 Access restricted
πŸ“Š Citations: 0


πŸ“„ Article: Astragalin activates autophagy and inhibits apoptosis of astrocytes in AD mice via down-regulating Fas/Fasl-VDAC1 pathway
πŸ‘₯ Authors: S. Wang, Shuhan; Y. Yang, Yaqi; J. Lin, Jiahong; … X. Jin, Xiaobao; Y. Ma, Yuxin
πŸ“š Journal: Free Radical Biology and Medicine, 2025
πŸ”— Full text: 🚫 Access restricted
πŸ“Š Citations: 0


πŸ“„ Article: Astragalin inhibits neuronal excitability and activates neuronal autophagy in the ACC and LH of CFA mice to alleviate inflammatory pain and pain-related emotions
πŸ‘₯ Authors: J. Lin, Jiahong; W. Zhang, Weishan; S. Wang, Shuhan; … X. Jin, Xiaobao; Y. Ma, Yuxin
πŸ“š Journal: International Immunopharmacology, 2025
πŸ”— Full text: 🚫 Access restricted
πŸ“Š Citations: 0